9-2-2　ⓔ文献

  1. Ehret GB, Teresa Ferreira, et al: The genetics of blood pressure regulation and its target organs from association studies in 342, 415 individuals. Nat Genet, 2016; 48, 1171–1184.

  2. Watanabe Y, Metoki H, et al: Accumulation of common polymorphisms is associated with development of hypertension: a 12–year follow–up from the Ohasama study. Hypertens Res, 2010; 33: 129–134.

  3. Xiao L, do Carmo LS, et al: Sympathetic enhancement of memory T–cell homing and hypertension sensitization. Circ Res, 2020; 126: 708–721.

  4. Ueda K, Nishimoto M, et al: Renal dysfunction induced by kidney–specific gene deletion of Hsd11b2 as a primary cause of salt–dependent Hypertension. Hypertension, 2017; 70: 111–118.

  5. Shibata S, Mu S, et al: Rac1 GTPase in rodent kidneys is essential for salt–sensitive hypertension via a mineralocorticoid receptor–dependent pathway. J Clin Invest, 2011; 121: 3233–3243.

  6. Mu S, Shimosawa T, et al: Epigenetic modulation of the renal beta–adrenergic–WNK4 pathway in salt–sensitive hypertension. Nat Med, 2011; 17: 573–580.

  7. Morris RC, Schmidlin O, et al: Vasodysfunction that involves renal vasodysfunction, not abnormally increased renal retention of sodium, accounts for the initiation of salt–induced hypertension. Circulation, 2016; 133: 881–893.